Abstract
Background: Sphingosine-1-phosphate (S1P) is a sphingolipid stored and released primarily by platelets, dependent on shear stress. Its effects on the endothelium are controlled by S1P receptors: S1P1 is probably the most important in arteries, activating the pathway reducing permeability. The combined effects of shear, flow and reactions on activation of S1P1 where simulated in a simplified arterial branch.
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