Abstract
Several lines of research recently pointed to- ward the identification of minor structural alter- ations and nucleotide substitutions that together are responsible for the 30% of mutations among patients affected with the X-linked Duchenne muscular dystrophy (DMD) or Becker muscular dystrophy (Bulman et al., 1991; Clemens et al.,
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have