Abstract

The resistance of human immunodeficiency virus type 1 (HIV-1) to antibody-mediated immunity often prevents the detection of antibodies that neutralize primary isolates of HIV-1. However, conventional assays for antibody functions other than neutralization are suboptimal. Current methods for measuring the killing of virus-infected cells by antibody-dependent cell-mediated cytotoxicity (ADCC) are limited by the number of natural killer (NK) cells obtainable from individual donors, donor-to-donor variation, and the use of nonphysiological targets. We therefore developed an ADCC assay based on NK cell lines that express human or macaque CD16 and a CD4(+) T-cell line that expresses luciferase from a Tat-inducible promoter upon HIV-1 or simian immunodeficiency virus (SIV) infection. NK cells and virus-infected targets are mixed in the presence of serial plasma dilutions, and ADCC is measured as the dose-dependent loss of luciferase activity. Using this approach, ADCC titers were measured in plasma samples from HIV-infected human donors and SIV-infected macaques. For the same plasma samples paired with the same test viruses, this assay was approximately 2 orders of magnitude more sensitive than optimized assays for neutralizing antibodies-frequently allowing the measurement of ADCC in the absence of detectable neutralization. Although ADCC correlated with other measures of Env-specific antibodies, neutralizing and gp120 binding titers did not consistently predict ADCC activity. Hence, this assay affords a sensitive method for measuring antibodies capable of directing ADCC against HIV- or SIV-infected cells expressing native conformations of the viral envelope glycoprotein and reveals incomplete overlap of the antibodies that direct ADCC and those measured in neutralization and binding assays.

Highlights

  • The resistance of human immunodeficiency virus type 1 (HIV-1) to antibody-mediated immunity often prevents the detection of antibodies that neutralize primary isolates of HIV-1

  • This assay affords a sensitive method for measuring antibodies capable of directing antibody-dependent cell-mediated cytotoxicity (ADCC) against HIV- or simian immunodeficiency virus (SIV)-infected cells expressing native conformations of the viral envelope glycoprotein and reveals incomplete overlap of the antibodies that direct ADCC and those measured in neutralization and binding assays

  • Of CD16ϩ cytolytic natural killer (NK) cells in the peripheral blood of these species (28, 107). This comparison demonstrates that the levels of CD16 expressed on the cloned NK cell lines are similar to or slightly lower than the levels expressed on the CD16ϩ populations of primary human and macaque NK cells

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Summary

Introduction

The resistance of human immunodeficiency virus type 1 (HIV-1) to antibody-mediated immunity often prevents the detection of antibodies that neutralize primary isolates of HIV-1. The constant (Fc) region of IgG interacts with Fc receptors expressed on leukocytes and with complement These interactions can contribute to antiviral immunity by inactivating and clearing virions (1, 121), orchestrating the homing of effector cells (37, 42, 56, 78, 90, 93, 94, 98, 99, 113, 131), inhibiting virus replication (23, 31, 33, 37, 45, 55, 70, 98, 128), and killing virus-infected cells by complement-dependent cytotoxicity (CDC) (120) or by antibody-dependent cell-mediated cytotoxicity (ADCC) (71, 75, 112). ELISAs using recombinant forms of Env and neutralization assays using T-cell lineadapted viruses measure antibodies that may not belong to the November 2012 Volume 86 Number 22 These drawbacks apply to current methods for measuring antibodies that direct ADCC. ADCC assays based on target cells coated with recombinant forms of Env or chronically infected T-cell lines measure antibodies that may not direct ADCC against cells infected with primary isolates

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