Abstract
Many β-lactamases classified by their amino acid sequences and functional substrate specificity profiles in various Gram-negative bacilli such as Pseudomonas spp. and members of the family Enterobacteriaceae have been documented.1 Since the late 1980s, extended-spectrum β-lactamases (ESBLs) derived from TEM- and SHV-type penicillinases capable of hydrolysing the oxymino-cephalosporins, have been spreading globally, mainly in Enterobacteriaceae, including Klebsiella pneumoniae and Escherichia coli.
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