Abstract
Group B streptococcus (Streptococcus agalactiae) is proposed as causative agent for the development of the sporadic form of Alzheimer‘s disease. Using a fibrinogen binding protein, aggregates are formed including A-beta. After triggering Alzheimer‘s disease by the bacterium, the next down-stream events mainly follow the well known so called A-beta hypothesis. The combination of the two hypotheses is able to explain a number of epidemiological and biochemial aspects of Alzheimer‘s disease.
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