Abstract
Spasticity is a motor disorder characterized by a velocity-dependent increase in tonic stretch reflexes [1], commonly seen in many neurological disorders. Clinically, spasticity is measured by an examiner rotating a joint and simultaneously estimating the resistance according to an ordinal scale. However, the limited reliability of the measurement and the impossibility to discriminate between the underlying neural (stretch reflex) and non-neural (i.e. muscle mechanics) contributions have been the motivation to develop methods describing resistance joint torque quantitatively. The aim of this preliminary study is to develop a forward neuromusculoskeletal model consisting of the explicit musculotendon, muscle spindle, and motoneuron pool, which can simulate the passive isokinetic ankle dorsiflexion test of spasticity.
Highlights
Spasticity is a motor disorder characterized by a velocitydependent increase in tonic stretch reflexes [1], commonly seen in many neurological disorders
The musculoskeletal geometry was based on the anthropometrical data from a healthy female
Activation dynamics were modeled as a first order differential equation
Summary
Spasticity is a motor disorder characterized by a velocitydependent increase in tonic stretch reflexes [1], commonly seen in many neurological disorders. Spasticity is measured by an examiner rotating a joint and simultaneously estimating the resistance according to an ordinal scale. The limited reliability of the measurement and the impossibility to discriminate between the underlying neural (stretch reflex) and non-neural (i.e. muscle mechanics) contributions have been the motivation to develop methods describing resistance joint torque quantitatively. The aim of this preliminary study is to develop a forward neuromusculoskeletal model consisting of the explicit musculotendon, muscle spindle, and motoneuron pool, which can simulate the passive isokinetic ankle dorsiflexion test of spasticity
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