Abstract
Klinefelter Syndrome (KS) is the most common genetic condition cause of non-obstructive azoospermia (NOA). KS can often result in decreased testicular growth and testosterone production. Exogenous testosterone therapy is commonly prescribed for KS patients to treat hypogonadism, though the exogenous testosterone therapy may have additional impacts to future fertility potential. Current clinical practice is to have KS adolescent patients provide frequent semen samples to identify potential sperm for early cryopreservation.
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