Abstract
Rationale: Low carbohydrate high fat diets (LCHFDs) are recommended for cancer patients with deranged carbohydrate metabolism. However, our recent studies demonstrated an LCHFD to accelerate cancer growth in a murine carcinomatous peritonitis (CP) model. Thus, LCHFDs might impair host immunity not only to cancer but also to microbial infection. Here, we examined influences of an LCHFD on effector sites of gut associated lymphoid tissue (GALT) (LP: lamina propria, IE: intraepithelial space) in the above mentioned CP model.
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