Abstract
Some of the first glimpses into acute pancreatitis were by Reginald H. Fitz, pathologist, who described the clinical features of acute pancreatitis in 1889 and subtyped it into hemorrhagic, suppurative, and gangrenous pancreatitis.1 However, he believed pancreatitis was the result of gastroduodenitis. In 1896, Chiari put forth the premise that pancreatitis was the result of pancreatic autodigestion.2 Claude Bernard demonstrated that pancreatic secretion was involved in the digestion of protein, carbohydrate, and fat in dogs.
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