Abstract
The inflammatory gingival hyperplasia is considered a non-neoplasic reactional proliferative process usually related to low intensity chronic irritative factors. Thus, the presence of periodontal diseases may lead to the development of this gingival hyperplasia by the presence of calculus and dental biofilm and to the gingival infection or inflammatory processes itself. The purpose of this paper is to review the possible ethiopathogenic mechanisms of this relationship as well as the clinical and histopathologic characteristics incidence and frequency differential diagnosis and treatment modalities applied on the inflammatory gingival hyperplasia.
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