Abstract

Factors relating to the occurrence of mastitis were studied on 12 Irish dairy herds with histories of elevated somatic cell count (SCC) and/or increased incidence of clinical mastitis cases. Milk recording data were analysed, housing conditions and calving areas were examined; dry cow therapy, clinical mastitis records, milking technique and aspects of milking machine function were assessed.Herds with a ratio of less than 110 cubicles per 100 cows were more likely to experience environmental mastitis. Herds with inadequate calving facilities, where cows spent prolonged periods on straw bedding, were likely to acquire environmental mastitis. In the majority of the herds, the selection of dry cow therapy lacked adequate planning. The majority of farmers took no action to reduce pain experienced by cows suffering mastitis. Deficiencies in parlour hygiene were evident in all herds experiencing elevation in SCC.

Highlights

  • Bovine mastitis may be classified as contagious or environmental

  • Streptococcus uberis is environmental in origin, it can be spread from cow to cow in a manner similar to that for contagious mastitis organisms

  • Seven of the herds were exclusively spring-calving, while the remaining five herds calved cows in both autumn and spring. This calving distribution would not accurately reflect the national situation where approximately 90% of Irish cows calve in the spring months (Anon, 2002a)

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Summary

Introduction

Bovine mastitis may be classified as contagious or environmental. The main contagious pathogens are Staphylococcus aureus and Streptococcus species. Streptococcus uberis is environmental in origin, it can be spread from cow to cow in a manner similar to that for contagious mastitis organisms. The mammary gland and udder skin act as reservoirs of infection in contagious mastitis. Infection is acquired during udder preparation or during the milking process. The presence of carrier cows in the herd, inappropriate

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