Abstract

Racemose and intraventricular neurocysticercosis are uncommon types of neurocysticercosis, resulting in a multiloculated, grape-like cluster appearance in the cerebrospinal fluid (CSF) spaces. A male patient presented with symptoms of raised intracranial pressure and demonstrated racemose neurocysticercosis at an atypical location involving the region of the crus of the fornix at the level of the body of lateral ventricles on magnetic resonance imaging. Associated intraventricular neurocysticercosis was seen in the atrium of the left lateral ventricle and fourth ventricle.

Highlights

  • Neurocysticercosis is the most common parasitic infection of the central nervous system

  • Cysticerci are located in basal cisterns, subarachnoid spaces and ventricles producing multiloculated cystic lesions with mass effect and a surrounding inflammatory reaction.[1]

  • This report describes an unusual location of racemose neurocysticercosis located anterior and inferior to the splenium of the corpus callosum and between the fornixes associated with intraventricular neurocysticercosis in the atrium of the left lateral ventricle and fourth ventricle

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Summary

Introduction

Neurocysticercosis is the most common parasitic infection of the central nervous system. Magnetic resonance imaging revealed multiple small clustered thin-walled cystic lesions in the region of crus of fornix at the level of the body of lateral ventricles. Multiple similar small clustered cysts were noticed in the atrium of the left lateral ventricle with enhancement of the cyst walls on post-contrast T1W images (Figure 2). A few thin membranes or septations were seen in the fourth ventricle on the 3-D spoiled gradient recalled echo (SGPGR BRAVO) T1 post-contrast sequence, obstructing the outlet of the fourth ventricle (Figure 3c). These intraventricular membranes were not well delineated on other conventional sequences. The possibility of an intraventricular neurocysticercosis cyst or post-inflammatory membrane was considered for the fourth ventricle lesion causing upstream obstruction. The patient was treated with albendazole and steroids and follow-up imaging was advised

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Conclusion
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