A BIOMECHANICAL STUDY OF THE EFFECTS OF FLEXION ANGLE ON THE INDUCTION MECHANISM OF CERVICAL SPONDYLOSIS

Abstract

Lesions in facet joints such as bone hyperplasia and degenerative changes in the intervertebral discs, can compress nerve roots and the spinal cord, leading to cervical spondylosis (CS). Lesions in these parts of the spine are commonly related to abnormal loads caused by bad posture of the cervical spine. This study aimed to understand the potential mechanical effects of load amplitude on cervical spine motion to provide a theoretical basis for the biomechanical causes of CS, and to provide a reference for preventing of the condition. In this study, a finite element model of the normal human cervical spine (C1-C7) was established and validated using an infrared motion capture system to analyze the effects of flexion angle on the stresses experienced by intervertebral discs, the anterior edge of the vertebral body, the pedicle, uncinate and facet joints. Our analysis indicated that the intervertebral disc load increased by at least 70% during the 20∘ to 45∘ flexion of the neck with 121% load increase in the vertebrae. In the intervertebral discs, the stress was largest at C4-C5, and the stress was moderate at C5-C6. These results are consistent with clinical CS prone site research. According to Wolff’s law, when bones are placed under large stresses, hyperplasia can result to allow adaptation to large loads. Increased cervical spine flexion angles caused the proliferation of bone in the above-mentioned parts of the spine and can accelerate accelerating the appearance of CS.