Abstract
Objective: Bacterial signal peptides are known to trigger innate immunity responses by activation of formyl peptide receptors (FPRs) present in immune cells, e.g. leukocytes. Members of the FPR-family are also found in the murine vomeronasal organ where they are candidates for chemosensory recognition of bacterial pathogens. Here, we investigated the effects of bacterial signal peptides on mucociliary clearance in the murine trachea.
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