Abstract
RENAL LESIONS associated with dysproteinemias and lymphoproliferative disorders are well known and include amyloid, cast nephropathy, monoclonal immunoglobulin deposition disease, cryoglobulin-associated glomerular disease, and fibrillary-immunotactoid glomerulopathy.1 When a patient with a monoclonal gammopathy has nephrotic-range proteinuria, one of the aforementioned renal lesions is often considered first in the differential diagnosis. However, more common secondary causes of heavy proteinuria in adults must still be entertained despite knowledge of the patient’s dysproteinemia.
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