Abstract
Homologous recombination deficiency (HRD) refers to the state of tumor cells which received defects in DNA-damage repair mechanisms. The HRD phenotype encodes important proteins for DNA homologous recombination repair (HRR) which can serve as a biomarker of therapeutic efficacy. Here, we explored the frequency and clinical significance of HRD gene mutations in non-cutaneous melanoma to provide experience for clinical options.
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