Abstract
Purpose: During environmental heat exposure in humans blood is dis- tributed to the compliant skin vasculature to facilitate heat loss. This causes peripheral venous pooling, decreases venous return and may com- promise cardiac output. Competition for limited maximal cardiac output therefore exists between the skin, to subserve thermoregulatory demands, and central blood pressure regulation. No previous studies have examined the impact of heating on skin blood flow (SkBF) responses in patients with heart failure (HF).
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