Abstract

Recent advances in the molecular biology of hepatocellular carcinoma (HCC) have identified new therapeutic targets that may potentiate the effects of transarterial embolization (TAE). Development of these new therapies requires a pre-clinical model that recapitulates the human disease and allows minimally invasive TAE that mimics the protocol used in patients. We describe a minimally invasive, superselective TAE in a translational rat model of HCC.

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