Abstract
The risk of central nervous system oxygen toxicity (CNSOT) limits diving operations for US Navy and occupational divers. CNSOT has variable and potentially deadly consequences such as seizures while underwater. Its pathophysiology is incompletely understood and its only current mitigation strategy is adhering to oxygen partial pressure (PO2) and time limits. Nutritional ketosis (NK) is used in the treatment of refractory epilepsy, and animal studies suggest it may delay onset of CNSOT. Primary objectives of this analysis are to better understand the pathophysiology and manifestations of CNSOT, and identify possible practical warning methods.
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