Abstract
There are considerable differences in the central processing of somatic and visceral pain and little is known about the role of the rostral ventromedial medulla (RVM) in descending modulation of visceral hyperalgesic states. In the present experiments we have investigated the responses of RVM “on” and “off” cells to a visceral noxious stimulus – intracolonic (IC) instillation of capsaicin (CAP) – and compared them with the responses to intradermal (ID) injection of CAP in their somatic receptive fields.
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