Abstract
Keloids are benign fibroproliferative tumors with features of raised scar tissue and excessive growth beyond the original border. Keloid scars show excessive extracellular matrix(ECM) deposition and rich vasculature. Recently, many studies have revealed the important pathways of keloid pathogenesis. Herein, we focus on the difference between traumatic and nontraumatic keloids in a transcriptomic level. To preclude intra-lesional heterogeneity, whole tissue was used for bulk RNA-sequencing. Three trauma induced keloids and 4 non-trauma related keloids were achieved by surgical excision from 7 different patients.
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