Abstract
Objective To demonstrate electroencephalographic evolution in catatonia, secondary to carbon monoxide poisoning. Case report A 28-year-old female, with accidental carbon monoxide exposition, showed drowsiness and motor focal seizures in right limbs, acutely. After 3 weeks, she presented with headache, memory dysfunction, and catatonic syndrome. Brain MRI showed bilateral frontoparietal hyper intensities in T2 and FLAIR with left predominance and associated restricted diffusion. Electroencephalographic evolution: The first EEG (after seizures) showed PLEDs, characterized by biphasic waves in left frontocentral region with an inter-discharge interval of 0.5–1 s. Second EEG (a week later, receiving treatment with amantadine and lorazepam), showed diffuse slowing without PLEDs. Third EEG, one month after the exposure, and complete clinical recovery, was normal. Comment The EEG in a patient with monoxide carbon and catatonia can show nonspecific findings, showing the most significant changes in the acute phase of the intoxication, and it proves to be useful in the differential diagnosis of catatonia.
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