Abstract
(Hgb 10 g/dL) in the absence of bleeding, elevated LDH, and undetectable haptoglobin ( 8g/dL). Endpoints analyzed were mortality, hospitalization, and need for transfusion. Results: From March 2010 – present, 12/84 (14.3%) HMII patients developed hemolysis. Among these, 1-year survival was 19.3%, lower than expected for supported HMII patients (Fig 1). Of the survivors, 2 had reversible causes of hemolysis, 1 was transplanted, and 2 are currently awaiting transplant at UNOS status 1A. In the 2 reversible cases, LDH was modestly elevated ( 500 units/L). In contrast, the other 10 patients had LDH values of 1700 units/L. Four of these 10 pumps were available for examination post-explant, and each had extensive thrombosis. In nonsurvivors, there were statistical trends toward higher LDH, higher bilirubin, and lower INR when compared to survivors. Blood transfusions and rehospitalization for hemolysis occurred in 11/12 patients. Interestingly, LVAD doppler velocities were normal in all but 2 patients where the velocities were 1.75 and 2.1 m/s.
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