Abstract
Staphylococcus aureus (S.aureus) plays a crucial role in the pathogenesis of atopic dermatitis (AD) via the secretion of several virulence factors that can exacerbate the disease. S.aureus growth and virulence inhibition is one of the potential therapeutic approaches in AD treatment. Ultraviolet radiation (UVR)-based phototherapy is one of the widely used treatment options for AD with a favourable benefit-risk ratio. UVR has been shown being able inducing antimicrobial peptides (AMP). We therefore hypothesize that the therapeutic efficacy of UVR is at least partly due to its ability to induce AMP secretion not only from the host cells, but also from certain skin resident microbes.
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