Abstract
Iron is an essential nutrient that functions as a cofactor for cellular enzymatic redox reactions. Hosts and pathogens compete over iron for survival. Previous studies have implicated the skin as a unique site for iron homeostasis as the major contributing source to iron excretion from the body: Primary human keratinocytes store iron as ferritin as a function of exogenous iron addition, with modulation of their differentiation. Wildtype mice subjected to systemic iron bolus or iron chelation demonstrate changes in total epidermal elemental iron content.
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