Abstract
The aim of this study was to identify at birth possible risk factors for neonatal hyperbilirubinemia (arbitrarily defined as a maximum serum bilirubin level 10 mg/dl in the first week of life) in infants compatible with their mothers in both the Rh and ABO system. A set of 21 genetic, developmental and environmental maternal and neonatal variables have been analysed in 162 consecutively born infants of both sexes. In males, cord bilirubin level, haptoglobin phenotype, anesthesia in labor and birth-weight contributed significantly to the separation of infants with hyperbilirubinemia. In females, maternal ABO blood-group, gestational lenght, cord bilirubin level, anesthesia in labor, Acid Phosphatase-1 and Phosphoglucomutase-1 phenotypes and smoke during pregnancy significantly contributed to the same separation. A group of infants with less than 1% risk of hyperbilirubinemia, including 25% of males and 40% of females, could be separated at birth using the above listed variables. Our results suggest that there are differences between males and females in the relations among the variables considered and in their predictive value for the development of hyperbilirubinemia.(Supported by CNR, MPI and Regional Funds).
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