Abstract
Background: The extensive involvement of interleukin enhancer binding factor 2 (ILF2) in RNA stability and inflammation response is well documented. Aberrant LncRNAs expression contributes to psoriasis development and progression. However, little is known about the role of ILF2 in psoriasis. Objectives: Aimed to explore the role of ILF2 and KLHDC7B-DT in psoriasis. Methods: Long noncoding RNA (lncRNA) expression in psoriatic tissues was measured by lncRNA microarray and qRT-PCR. Normal human epidermal keratinocytes (NHEKs), HaCaT cells, and Ker-CT cells stimulated by M5 (IL-17A, IL-22, IL-1 α, oncostatin M, and TNF-α) were used to establish a psoriasis model in vitro.
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