Abstract
Introduction: Atrial fibrillation (AF) is a common arrhythmia associated with poor outcomes. N-3 fatty acids have been shown to provide significant cardiovascular risk reduction, but they may exacerbate the risk of AF. The pathway by which n-3 fatty acids may be arrhythmogenic is unknown. Both left atrial (LA) size and left ventricular (LV) mass have been cited as risk factors for development of AF. One possible mechanism by which n-three fatty acids may increase AF risk involves cardiac chamber morphology alteration.
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