Abstract
Atopic dermatitis (AD) is a common, pruritic, inflammatory skin disease characterized by life-long periods of acute disease flares and remissions. Predisposition to AD is associated with genetic variation. Killer cell Ig-like receptor family (KIR) receptors interact with HLA ligands and are thought to be a primary regulator of NK cell function. NK cells have also been implicated in the pathophysiology of AD and other skin inflammatory diseases. In this study, we examined allelic variation in KIR genes KIR2DL5, KIR2DS5, and KIR2DS1 with respect to AD.
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