Abstract
Fetal alcohol syndrome (FAS) is associated with mental retardation and neurodevelopmental abnormalities. Neuroprotective peptides (NAP+SAL) can prevent some of the alcohol-induced damage including fetal death, growth abnormalities and learning impairment in part through preventing alcohol-induced alterations in NMDA gene expressiON Given the role of cytokines and chemokines on NMDA receptors, we evaluated a panel in a model of FAS to evaluate their role and if the neuroprotective peptides NAP+SAL work through preventing these alterations.
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