Abstract
Impaired nitric oxide-mediated pulmonary vascular tone is commonly found in SHF and is associated with changes in LV hemodynamics and decreased exercise capacity. PDE5 inhibition can be helpful in this setting. We investigated the effects of udenafil, a new and long-acting PDE5 inhibitor, on left ventricular (LV) hemodynamics and exercise capacity in systolic heart failure (SHF).
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