Abstract
Atopic dermatitis (AD) is a widespread chronic inflammation of the skin with a diverse and complex pathogenesis. AD is characterized by a dysregulation of barrier molecules such as keratins and filaggrin (FLG) including FLG mutations. In addition, a dysbiosis of the cutaneous microbiota is associated with AD. However, it is still not completely understood whether the dysbiosis of the cutaneous microbiota is a cause or a consequence of the destroyed integrity of the skin barrier in AD. Functional approaches to address this question are rare.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
