Abstract
The glutamate hypothesis of schizophrenia highlights disturbances in the N-methyl-D-aspartate receptor (NMDAR) glutamate-gated cation channel, and there have been increasing interests in this receptor as a new target for drugs. A large body of studies reported increased glutamate concentrations in patients with schizophrenia specifically in the frontal region; however, there have been limited reports in high-risk adolescents for psychosis (HR).
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Schedule a callMore From: Journal of the American Academy of Child & Adolescent Psychiatry
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