Abstract
UV exposure is known to cause skin irritation, erythema, darkening and disrupted barrier, partially through inducing oxidative stress and inflammation. Glutathione is a major antioxidant in skin to maintain the redox balance and protect against stress. Previous studies suggested glutathione amino acids precursors (GAPs) effectively boosted cellular glutathione and provided protection from UV and oxidative stress-induced skin damage and darkening. The current study aimed to evaluate the in vitro and in vivo protection of GAPs under UV.
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