Abstract
Atopic dermatitis (AD) is a chronic inflammatory disorder with the highest disease burden amongst skin diseases and a significant unmet need to improve treatment. Differences in microbiota composition and increased abundance of Staphylococcus aureus have been observed in the skin of AD patients in contrast with healthy individuals. Tryptophan (Trp) metabolites have been shown to have strong antimicrobial activity against S. aureus and current data suggest that concentrations of these metabolites are depleted in AD skin.
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