Abstract
Resident memory T cells (TRM) are a critical component of the tissue immune response but are also implicated in the physiopathology of cutaneous chronic inflammatory diseases, such as vitiligo. In this depigmenting disease characterized by the loss of epidermal melanocytes, the involvement of skin TRM with a type-1 skewed immune response (production of IFNγ and TNFα) in lesions is commonly admitted. Our recent data suggest an additional role of a type-2 immune response with local production of IL-13.
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