Abstract
Natural history of cystic fibrosis (CF) is marked by an increasing proneness to lung infections requiring long-term antibiotic therapy. Secretory IgA (SIgA) represent a major component of the mucosal defence system but they have been shown to be ineffective to prevent lung colonization from Pseudomonas Aeruginosa in CF. Little is known however about secretory immunity to common bacteria in CF patients.
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