Abstract
Development of BOS following human lung transplantation (LTx) is a major limiting factor in the long term survival of the lung allografts. Respiratory viral infections (RVI) following human LTx have been shown to be a significant risk factor for development of BOS. However the mechanisms by which RVI induce development of BOS remains undefined. The objective of this study was to determine the mechanisms by which RVI induce development of BOS.
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