Abstract
Prematurity is associated with fetal neuroinflammation and subsequent neurologic injury. The impact of magnesium sulfate (MgSO4) and betamethasone (BMTZ), commonly employed therapies in the setting of preterm labor, on prematurity-related neurologic morbidity remains incompletely understood. The objective of this study was to identify effects of MgSO4 and BMTZ on markers of inflammation and glial development in offspring of a murine model of preterm birth and perinatal brain injury.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
