Abstract
respectively). An increased production of IFN-gamma was typical of both Vdelta1 and Vdelta2 in non responders CHC. No differences were found in IL-17 and TGF-beta production. Conclusion: A selectively enriched and activated peripheral Vdelta1 population characterises HCV patients with advanced, more active and long-lasting infection; its enhanced IFN-gamma production may explain, possibly via the overxpression of IFN-gammainducible protein-10, non-response to antiviral therapy.
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