Abstract
236 SPL84, an antisense oligonucleotide modulating the 3849+10 kb C-T splicing defect, efficiently penetrates and restores CFTR function when treated through the apical side of primary human bronchial epithelial cells
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Published version (
Free)
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
