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Abstract
Purpose Fluid shear stress was thought to lead to atherosclerotic plaque progression, but such measurement procedures are often invasive. The goal of the present study is to address the biomechanical pathways of atherosclerosis progression and regression with our non-invasive MRI methods and a 3D fluid-structure interaction (FSI) model. The study was performed in a large animal atherosclerotic model.
Highlights
Jie Zheng*1, Dana R Abendschein1, Ruth J Okamoto1, Deshan Yang1, Kyle S McCommis1, Bernd Misselwitz2, Robert J Gropler1 and Dalin Tang3
Abstracts of the 11th Annual SCMR Scientific Sessions - 2008 Meeting abstracts – A single PDF containing all abstracts in this Supplement is available here. http://www.biomedcentral.com/content/pdf/1532-429X-10-S1-info.pdf
The goal of the present study is to address the biomechanical pathways of atherosclerosis progression and regression with our non-invasive MRI methods and a 3D fluid-structure interaction (FSI) model
Summary
Jie Zheng*1, Dana R Abendschein, Ruth J Okamoto, Deshan Yang, Kyle S McCommis, Bernd Misselwitz, Robert J Gropler and Dalin Tang. Abstracts of the 11th Annual SCMR Scientific Sessions - 2008 Meeting abstracts – A single PDF containing all abstracts in this Supplement is available here. http://www.biomedcentral.com/content/pdf/1532-429X-10-S1-info.pdf . Fluid shear stress was thought to lead to atherosclerotic plaque progression, but such measurement procedures are often invasive. The goal of the present study is to address the biomechanical pathways of atherosclerosis progression and regression with our non-invasive MRI methods and a 3D fluid-structure interaction (FSI) model. The study was performed in a large animal atherosclerotic model
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