Abstract
Publisher Summary Thyroid stimulating hormone (TSH) stimulates a variety of metabolic and morphologic parameters in thyroid cells. TSH initially attaches to specific receptors on the cell membrane and subsequently activates the adenylate cyclase-cyclic AMP system. Most of the morphologic and metabolic effects of TSH including stimulation of glucose oxidation are probably mediated by cyclic AMP, but this is less well established for 32 P incorporation into phospholipids. Although the exact biochemical steps involved in TSH stimulation of glucose oxidation and phospholipid synthesis have not been elucidated, measurement of these two effects provides a convenient assessment of the hormone's action. However, neither of these effects is specific for TSH and inhibition of them does not necessarily indicate abolition of other actions of TSH.
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