Abstract
Apoptotic mechanisms are thought to be important in wound healing for the removal of inflammatory cells and evolution of granulation tissue into scar. However, little is understood about the signal propagation and cellular mechanisms responsible for triggering programmed cell death in tissue injury particularly their involvement in fetal wound repair. Understanding these biochemical signals may lead to insights regarding scarless wound healing. In this study, we hypothesized that differences in cellular apoptosis would exist in mid- (E15) compared to late-gestational (E18) fetal mice subjected to cutaneous wounds.
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