Abstract
Atherosclerosis is a leading cause of morbidity and mortality within Canada. ‘Vulnerable' rupture prone plaque is associated with pronounced inflammatory activity and increased density of macrophage cells. Macrophages have increased metabolic rates and require high-levels of energy for phagocytic activity. Therefore, the uptake of radiolabelled glucose or [18F]-fluorodeoxyglucose (18FDG) imaged with hybrid positron emission tomography (PET) and computed tomography (CT) may serve as a surrogate marker of inflammatory activity within plaque.
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