Abstract
Study Objectives: Chest pain is one of the most common entrance complaints in the emergency department (ED). Rapid diagnostic times involving all aspects of acute coronary syndrome care is a major therapeutic goal. Chest pain secondary to acute coronary syndrome is hypothesized to promote a transformation in blood serum composition (eg, markers of plaque rupture, inflammation, ischemia) that is reflected as characteristic changes (“fingerprints”) in the corresponding spectra of serum samples. The objective of this prospective cohort study is to discover and exploit these characteristic changes, and thus to develop a novel, rapid and sensitive diagnostic test to rule in/rule out acute coronary syndrome as the origin of chest pain in ED patients for whom EKG and traditional infarction markers are negative.
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