Abstract
The clinical consequences of trauma differ across individuals: while some develop post-traumatic stress disorder (PTSD), others may not present with significant psychopathology. A growing body of evidence is pointing to the anatomical and physiological properties of the fear conditioning circuitry, and the amygdala in particular, as the substrate that explains different trauma-related clinical trajectories. In this study we use validated atlas-based methods to assess differences in the volumes of key amygdala subnuclei across patients with PTSD, asymptomatic trauma-expose individuals and healthy controls.
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