Abstract
Pancreatic ductal adenocarcinoma (PDAC) which is one of the most aggressive forms of cancer, is the 7th leading cause of cancer death globally. Due to the complexities in its biology and management, PDAC has a 5-year survival of less than 8%. The T cell diversity in the immune microenvironment of PDAC has been studied by existing research, however, their origin and functional roles are yet to be deciphered. With the subpar performance of immunotherapies like checkpoint blockade, there is a necessity to improve our understanding of the temporal characterization of leukocytes in the PDAC microenvironment.
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