Abstract
Background: The immunopathogenesis of hidradenitis suppurativa (HS) involves an exaggerated response to ruptured follicles. T-cells and neutrophils are attracted to follicles and produce IL-12 and IL-23 leading to keratinocyte proliferation and cytokine production. Aberrant cytokine expression has been demonstrated in skin and serum of HS patients, specifically increased serum IL-6, TNF-α, IL-17, and IL-1β, and IL-12, IL-23, IL-17, and TNF-α in skin, among others. This study aimed to characterize cytokine profiles in HS.
Published Version
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