Abstract
PGD is a significant cause of early morbidity and mortality after lung transplantation and is characterized by severe hypoxemia and infiltrates in the allograft. The pathogenesis of PGD is unclear, however subclinical increases in pulmonary venous pressure from left ventricular (LV) diastolic dysfunction may contribute by increasing capillary leak. We hypothesized that a higher ratio of early mitral inflow velocity (E) to early diastolic mitral annular velocity (é), indicative of worse diastolic function, would be associated with a higher risk of PGD.
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